Which of the following is an expected outcome of ingesting AAS?

Which of the following is an expected outcome of ingesting AAS?

• A. Decrease in red blood cell count
• B. Increased strength
• C. Reduced muscle mass
• D. Enhanced fat synthesis

Answer: (B)

Ingesting anabolic-androgenic steroids (AAS) is commonly associated with an increase in strength. This outcome occurs because AAS enhance protein synthesis and muscle recovery, leading to greater muscle mass and improved performance during resistance training. The increase in muscle size and strength is a primary reason why athletes and bodybuilders choose to use these substances, as they can facilitate quicker progress in strength training and physical fitness. The other outcomes listed do not align with the effects typically associated with AAS use. For instance, a decrease in red blood cell count is not a typical result of AAS and does not reflect their usual impact on physiology, which often includes increased production of red blood cells, particularly with certain types of steroids that stimulate erythropoiesis. Similarly, AAS are well-known for promoting muscle mass, so reduced muscle mass would be contrary to their intended effects. Lastly, enhanced fat synthesis is also not a characteristic response to AAS; rather, they often promote changes in body composition that favor muscle gain over fat accumulation. Thus, the primary expected outcome of ingesting AAS is indeed the increase in strength.

Ingesting anabolic-androgenic steroids (AAS) is commonly associated with an increase in strength. This outcome occurs because AAS enhance protein synthesis and muscle recovery, leading to greater muscle mass and improved performance during resistance training. The increase in muscle size and strength is a primary reason why athletes and bodybuilders choose to use these substances, as they can facilitate quicker progress in strength training and physical fitness.

The other outcomes listed do not align with the effects typically associated with AAS use. For instance, a decrease in red blood cell count is not a typical result of AAS and does not reflect their usual impact on physiology, which often includes increased production of red blood cells, particularly with certain types of steroids that stimulate erythropoiesis. Similarly, AAS are well-known for promoting muscle mass, so reduced muscle mass would be contrary to their intended effects. Lastly, enhanced fat synthesis is also not a characteristic response to AAS; rather, they often promote changes in body composition that favor muscle gain over fat accumulation. Thus, the primary expected outcome of ingesting AAS is indeed the increase in strength.